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Disease Info Card
Liddle Syndrome
Information about Liddle Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Liddle Syndrome
Most recent studies have shown that Liddle Syndrome shares some biological mechanisms with adrenal-hyperplasia, alkalosis, bartter-disease, conn-syndrome, cortisol-11-beta-ketoreductase-deficiency, cystic-fibrosis, essential-hypertension, fibrosis, hyperaldosteronism, hyperplasia, hypertensive-disease, hypoaldosteronism, hypotension-adverse-event, kidney-diseases, metabolic-alkalosis, mineralocorticoid-excess-syndrome-apparent, pseudohypoaldosteronism, pseudohypoaldosteronism-type-1.
Among the many pathways, these few ones have gauged particular interests from scientists studying Liddle Syndrome, and have been seen in publications frequently: Aging, Aldosterone Secretion, Clathrin-mediated Endocytosis, Endocytosis, Excretion, Exocytosis, Glycosylation, Ion Transport, Localization, Pathogenesis, Proteolysis, Regulation Of Blood Pressure, Renal Potassium Excretion, Response To Amiloride, Response To Camp, Secretion, Transepithelial Transport, Transport, Water Homeostasis, Water Transport
Quite a number of genes have been found to play important roles in Liddle Syndrome, such as ACE, AGT, ASIC5, CFTR, CYP11B2, HSD11B2, NEDD4, NEDD4L, NR3C1, NR3C2, POMC, REN, SCNN1A, SCNN1B, SCNN1G, SLC12A3, UBE2K, UBR5. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.