Disease Info Card

Hyperaldosteronism

Information about Hyperaldosteronism: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Hyperaldosteronism

Most recent studies have shown that Hyperaldosteronism shares some biological mechanisms with adenoma, adrenal-cortical-adenoma, adrenal-gland-diseases, adrenal-gland-neoplasms, adrenal-hyperplasia, alkalosis, bartter-disease, conn-syndrome, cushing-syndrome, essential-hypertension, heart-failure, hyperplasia, hypertensive-disease, kidney-diseases, neoplasms, pheochromocytoma, renal-hypertension, secondary-hyperaldosteronism, secondary-hypertension, tumors-of-adrenal-cortex.

Among the many pathways, these few ones have gauged particular interests from scientists studying Hyperaldosteronism, and have been seen in publications frequently: Aging, Aldosterone Secretion, Cell Proliferation, Chloride Transport, Circadian Rhythm, Cortisol Secretion, Diuresis, Excretion, Glomerular Filtration, Hormone Secretion, Insulin Secretion, Localization, Natriuresis, Pathogenesis, Reflex, Regulation Of Aldosterone Secretion, Secretion, Transport, Vasoconstriction, Vasodilation

Quite a number of genes have been found to play important roles in Hyperaldosteronism, such as ACE, AGT, AMY2A, AVP, BLOC1S6, CYP11B1, CYP11B2, ENPEP, EPB42, INS, KLK4, NPPA, NR3C2, POMC, PRH1, PTH, REN, S100A6. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.