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- Table of Contents
Information about Metabolic Alkalosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Metabolic Alkalosis shares some biological mechanisms with acid-base-imbalance, acidosis, acidosis-respiratory, alkalosis, alkalosis-respiratory, bartter-disease, dehydration, diarrhea, gitelman-syndrome, hyperaldosteronism, hypercapnia, hypertensive-disease, hypochloraemia, kidney-diseases, kidney-failure, metabolic-acidosis, neoplasms, vomiting, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Metabolic Alkalosis, and have been seen in publications frequently: Acid Secretion, Aldosterone Secretion, Bicarbonate Transport, Bone Resorption, Chloride Transport, Coagulation, Diuresis, Excretion, Gastric Acid Secretion, Glomerular Filtration, Glycolysis, Intestinal Absorption, Ion Transport, Lactation, Localization, Natriuresis, Pathogenesis, Secretion, Transport, Vasoconstriction
Quite a number of genes have been found to play important roles in Metabolic Alkalosis, such as AGT, ALB, APC, AVP, C2, CFTR, CLCNKB, GLUL, INS, KCNJ1, POMC, PTH, PTRH1, RAPGEF5, REN, SLC12A1, SLC12A3, SLC26A4, TESC, TSC1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.