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Disease Info Card
Bartter Disease
Information about Bartter Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Bartter Disease
Most recent studies have shown that Bartter Disease shares some biological mechanisms with acidosis, alkalosis, complete-hearing-loss, cushing-syndrome, gitelman-syndrome, hyperaldosteronism, hypercalciuria, hyperplasia, hypertensive-disease, hypertrophy, kidney-diseases, kidney-failure, metabolic-alkalosis, nephrocalcinosis, polyhydramnios, polyuria, vomiting, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Bartter Disease, and have been seen in publications frequently: Aldosterone Secretion, Cation Transport, Cellular Localization, Chloride Transport, Circadian Rhythm, Cortisol Secretion, Diuresis, Excretion, Glomerular Filtration, Hormone Secretion, Ion Transport, Localization, Natriuresis, Pathogenesis, Platelet Aggregation, Secretion, Transepithelial Chloride Transport, Transepithelial Transport, Transport, Vasodilation
Quite a number of genes have been found to play important roles in Bartter Disease, such as AGT, ANG, APC, AVP, BLM, BSND, CASR, CLCNKA, CLCNKB, DBT, GLUL, GP1BB, KCNJ1, KLK4, POMC, PTH, REN, SLC12A1, SLC12A3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.