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Disease Info Card
Methylenetetrahydrofolate Reductase Deficiency
Information about Methylenetetrahydrofolate Reductase Deficiency: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Methylenetetrahydrofolate Reductase Deficiency
Most recent studies have shown that Methylenetetrahydrofolate Reductase Deficiency shares some biological mechanisms with 510-methylenetetrahydrofolate-reductase-deficiency, cardiovascular-diseases, cerebrovascular-accident, congenital-abnormality, congenital-heart-defects, developmental-delay-(disorder), encephalopathies, folic-acid-deficiency, homocystinuria, hyperhomocysteinemia, inborn-errors-of-metabolism, malignant-neoplasms, muscle-spasticity, nervous-system-disorder, pregnancy-complications, psychotic-disorders, thrombosis, vascular-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Methylenetetrahydrofolate Reductase Deficiency, and have been seen in publications frequently: Brain Development, Cell Development, Cell Maturation, Cell Proliferation, Cytokine Secretion, Demethylation, Dna Hypomethylation, Dna Methylation, Lactation, Long-term Memory, Mating, Methylation, Myelination, Neurogenesis, Pathogenesis, Reflex, Spermatogenesis, Transport, Transsulfuration, Vasoconstriction
Quite a number of genes have been found to play important roles in Methylenetetrahydrofolate Reductase Deficiency, such as ACHE, APOA1, BRCA2, CBS, CHAT, DHFR, FADD, FANCD2, FMR1, IL10, ITPR1, MBP, MTHFR, MTR, MTRR, PSEN1, RELN, S100A8, TNFAIP1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.