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- Table of Contents
Information about Folic Acid Deficiency: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Folic Acid Deficiency shares some biological mechanisms with 510-methylenetetrahydrofolate-reductase-deficiency, anemia, anemia-macrocytic, anemia-megaloblastic, anemia-pernicious, celiac-disease, congenital-abnormality, epilepsy, hyperhomocysteinemia, hypochromic-anemia, iron-deficiency, iron-deficiency-anemia, malabsorption-syndrome, malignant-neoplasms, malnutrition, neoplasms, pregnancy-complications, pregnancy-complications-hematologic, vitamin-b-12-deficiency.
Among the many pathways, these few ones have gauged particular interests from scientists studying Folic Acid Deficiency, and have been seen in publications frequently: Aging, Cell Cycle, Cell Death, Cell Proliferation, Coagulation, Cognition, Dna Hypomethylation, Dna Methylation, Dna Repair, Excretion, Intestinal Absorption, Lactation, Mating, Methylation, Pathogenesis, Regeneration, S Phase, Secretion, Transport, Transsulfuration
Quite a number of genes have been found to play important roles in Folic Acid Deficiency, such as ALB, CBS, CSF2, DHFR, EPO, FANCA, FOLR1, FXN, LAMC2, MTHFR, MTR, RFC1, SLC19A1, SLC46A1, TCN2, TF, TYMS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.