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Disease Info Card
Hyperhomocysteinemia
Information about Hyperhomocysteinemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hyperhomocysteinemia
Most recent studies have shown that Hyperhomocysteinemia shares some biological mechanisms with 510-methylenetetrahydrofolate-reductase-deficiency, arteriosclerosis, atherosclerosis, cardiovascular-diseases, cerebrovascular-accident, coronary-artery-disease, coronary-heart-disease, diabetes-mellitus, folic-acid-deficiency, heart-diseases, hypertensive-disease, infarction, kidney-diseases, kidney-failure-chronic, thromboembolism, thrombophilia, thrombosis, vascular-diseases, venous-thrombosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hyperhomocysteinemia, and have been seen in publications frequently: Aging, Cell Adhesion, Cell Death, Cell Proliferation, Coagulation, Cognition, Dna Hypomethylation, Dna Methylation, Excretion, Fibrinolysis, Glomerular Filtration, Hemostasis, Methylation, Pathogenesis, Platelet Activation, Platelet Aggregation, Secretion, Transport, Transsulfuration, Vasodilation
Quite a number of genes have been found to play important roles in Hyperhomocysteinemia, such as ALB, CBS, CCL2, CRP, F2, F5, HTT, INS, LPA, MMP9, MTHFR, MTR, MTRR, NDUFB3, NOS3, TNF, TNFAIP1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.