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Information about Irido-corneo-trabecular Dysgenesis (disorder): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Irido-corneo-trabecular Dysgenesis (disorder) shares some biological mechanisms with aniridia, cataract, congenital-abnormality, congenital-corneal-opacity, corneal-diseases, crest-syndrome, disorder-of-eye, dystrophy, eye-abnormalities, glaucoma, graft-failure, hydrophthalmos, hypoplasia, intraocular-pressure-disorder, irido-corneal-dysgenesis, microphthalmos, peters-plus-syndrome, primary-congenital-glaucoma, rieger-syndrome, tissue-adhesions.
Among the many pathways, these few ones have gauged particular interests from scientists studying Irido-corneo-trabecular Dysgenesis (disorder), and have been seen in publications frequently: Cell Cycle, Cell Differentiation, Cell Proliferation, Eye Development, Glycosylation, Localization, Oncogenesis, Palate Development, Pathogenesis, Regulation Of Cell Proliferation, Wound Healing
Quite a number of genes have been found to play important roles in Irido-corneo-trabecular Dysgenesis (disorder), such as CYP1B1, EXT1, FKRP, FOXC1, FOXE3, INVS, MYOC, PAX2, PAX6, PITX2, PITX3, RB1, RBL2, SLC4A11, SS18L1, VSX2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.
Cell Cycle | Cell Differentiation | Cell Proliferation |
Eye Development | Glycosylation | Localization |
Oncogenesis | Palate Development | Pathogenesis |
Regulation Of Cell Proliferation | Wound Healing |