This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Peters Plus Syndrome
Information about Peters Plus Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Peters Plus Syndrome
Most recent studies have shown that Peters Plus Syndrome shares some biological mechanisms with atrophy, cataract, cleft-lip, cleft-palate-with-cleft-lip, congenital-abnormality, congenital-disorders, corneal-diseases, developmental-delay-(disorder), developmental-disabilities, dwarfism, eye-abnormalities, growth-disorders, irido-corneo-trabecular-dysgenesis-(disorder), limb-deformities-congenital, microphthalmos, multicystic-dysplastic-kidney.
Among the many pathways, these few ones have gauged particular interests from scientists studying Peters Plus Syndrome, and have been seen in publications frequently: Glycosylation, Methylation, Pathogenesis
Quite a number of genes have been found to play important roles in Peters Plus Syndrome, such as BBS9, CFP, CYP1B1, FGFR2, INVS, LMLN, MS4A1, PAX6, PITX2, PITX3, ROR2, RORA, SPANXB1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.
Pathways Related to Peters Plus Syndrome
This information is being compiled and will come in a future update
| Glycosylation | Methylation | Pathogenesis |