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Disease Info Card
Graft Failure
Information about Graft Failure: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Graft Failure
Most recent studies have shown that Graft Failure shares some biological mechanisms with acute-gvh-disease, chronic-graft-versus-host-disease, corneal-diseases, decreased-immunologic-activity-[pe], diabetes-mellitus, graft-occlusion-vascular, graft-vs-host-disease, hemorrhage, hyperplasia, hypertensive-disease, infective-disorder, ischemia, kidney-diseases, kidney-failure-chronic, leukemia, malignant-neoplasms, stenosis, thrombosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Graft Failure, and have been seen in publications frequently: Angiogenesis, Cell Death, Cell Migration, Cell Proliferation, Coagulation, Dehiscence, Donor Selection, Excretion, Glomerular Filtration, Immune Response, Localization, Muscle Cell Proliferation, Pathogenesis, Regeneration, Secretion, Sensitization, Smooth Muscle Cell Proliferation, Translation, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Graft Failure, such as ABO, ACLY, BCR, CABIN1, CD34, CSF3, CTLA4, HLA-A, HLA-B, HLA-DQA1, HLA-DRB4, IL2, IL6, INS, MID1, NOD2, RAPGEF5, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.