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Disease Info Card
Rieger Syndrome
Information about Rieger Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Rieger Syndrome
Most recent studies have shown that Rieger Syndrome shares some biological mechanisms with aniridia, atrophy, congenital-abnormality, corneal-diseases, craniofacial-abnormalities, crest-syndrome, cytogenetic-abnormality, developmental-absence-of-tooth, disorder-of-eye, dysplasia, eye-abnormalities, glaucoma, hydrophthalmos, hypodontia, hypoplasia, intraocular-pressure-disorder, irido-corneal-dysgenesis, pituitary-diseases, tissue-adhesions, tooth-abnormalities.
Among the many pathways, these few ones have gauged particular interests from scientists studying Rieger Syndrome, and have been seen in publications frequently: Amelogenesis, Cell Adhesion, Cell Migration, Cell Proliferation, Eye Development, Eye Morphogenesis, Gland Development, Lacrimal Gland Development, Localization, Lung Development, Muscle Cell Differentiation, Myoblast Proliferation, Neural Crest Cell Migration, Neurogenesis, Odontogenesis, Ossification, Pathogenesis, Pigmentation, Pituitary Gland Development, Translation
Quite a number of genes have been found to play important roles in Rieger Syndrome, such as APCS, CYP1B1, EGF, FOXC1, HESX1, INS, PAX6, PBX1, PITX1, PITX2, PITX3, POU1F1, PRL, SHH, SLC20A1, SLURP1, SS18L1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.