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Disease Info Card
Irido-corneal Dysgenesis
Information about Irido-corneal Dysgenesis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Irido-corneal Dysgenesis
Most recent studies have shown that Irido-corneal Dysgenesis shares some biological mechanisms with aniridia, aphakia, blind-vision, cataract, congenital-abnormality, corneal-diseases, crest-syndrome, disorder-of-eye, dysplasia, eye-abnormalities, glaucoma, hydrophthalmos, hypoplasia, intraocular-pressure-disorder, irido-corneo-trabecular-dysgenesis-(disorder), microphthalmos, retinal-dysplasia, rieger-syndrome, tissue-adhesions.
Among the many pathways, these few ones have gauged particular interests from scientists studying Irido-corneal Dysgenesis, and have been seen in publications frequently: Angiogenesis, Cell Adhesion, Cell Cycle, Cell Death, Cell Differentiation, Cell Migration, Cell-matrix Adhesion, Coagulation, Eye Development, Fertilization, Gastrulation, Localization, Mesenchymal Cell Differentiation, Mesenchyme Development, Methylation, Nerve Development, Neural Crest Cell Migration, Optic Vesicle Formation, Pathogenesis, Wound Healing
Quite a number of genes have been found to play important roles in Irido-corneal Dysgenesis, such as ABCC6, BMP4, COL4A1, CYP1B1, F8, FOS, FOXC1, FOXE3, JUN, LGR4, MAF, PAX6, PITX2, PITX3, SS18L1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.