This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Classical Lissencephaly
Information about Classical Lissencephaly: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Classical Lissencephaly
Most recent studies have shown that Classical Lissencephaly shares some biological mechanisms with brain-diseases, congenital-abnormality, congenital-anomaly-of-brain, congenital-cerebellar-hypoplasia, congenital-neurologic-anomalies, dysmyelopoietic-syndromes, dysplasia, ectopic-tissue, epilepsy, hypoplasia, lissencephaly, lissencephaly-i, miller-dieker-syndrome, neuronal-migration-disorders, pachygyria, periventricular-nodular-heterotopia, subcortical-band-heterotopia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Classical Lissencephaly, and have been seen in publications frequently: Brain Development, Cell Division, Cell Migration, Cell Proliferation, Central Nervous System Development, Chromosome Breakage, Interphase, Localization, Meiosis, Mitosis, Nervous System Development, Neuroblast Migration, Neurogenesis, Pathogenesis, Prophase, Somal Translocation, Stem Cell Division, System Development, Translation, Transport
Quite a number of genes have been found to play important roles in Classical Lissencephaly, such as ARX, ASXL1, CDK5R1, DCLK2, DCX, DISC1, FEZ1, LZTS1, NDE1, NDEL1, NUDC, PAFAH1B1, PLA2G7, RELN, SF3B1, TMED9, TUBA1A, UBA2, YWHAE. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.