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Disease Info Card
Classical Lissencephalies And Subcortical Band Heterotopias
Information about Classical Lissencephalies And Subcortical Band Heterotopias: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Classical Lissencephalies And Subcortical Band Heterotopias
Most recent studies have shown that Classical Lissencephalies And Subcortical Band Heterotopias shares some biological mechanisms with classical-lissencephaly, congenital-anomaly-of-face, developmental-delay-(disorder), developmental-disabilities, ectopic-tissue, epilepsy, growth-retardation, intractable-epilepsy, lissencephaly, malformations-of-cortical-development, miller-dieker-syndrome, nervous-system-disorder, neuronal-migration-disorders, pachygyria, subcortical-band-heterotopia, x-linked-lissencephaly.
Among the many pathways, these few ones have gauged particular interests from scientists studying Classical Lissencephalies And Subcortical Band Heterotopias, and have been seen in publications frequently: Brain Development, Cell Cycle, Cell Division, Cell Proliferation, Eye Development, Microtubule-based Transport, Myelination, Neurogenesis, Pathogenesis, Regulation Of Growth, Rna Interference, Synaptic Transmission, Transport
Quite a number of genes have been found to play important roles in Classical Lissencephalies And Subcortical Band Heterotopias, such as ARX, CALB1, CALB2, CRK, DCX, EOMES, EXOSC1, NDEL1, OCRL, PAFAH1B1, PLA2G7, RELN, SF3B1, SST, TUBA1A, UBA2, YWHAE. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.