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Disease Info Card
X-linked Lissencephaly
Information about X-linked Lissencephaly: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of X-linked Lissencephaly
Most recent studies have shown that X-linked Lissencephaly shares some biological mechanisms with agenesis-of-corpus-callosum, ambiguous-genitalia, classical-lissencephalies-and-subcortical-band-heterotopias, congenital-absence, congenital-anomaly-of-brain, ectopic-tissue, epilepsy, hypoplasia, infantile-spasms, intractable-epilepsy, lissencephaly, microcephaly, miller-dieker-syndrome, neuronal-migration-disorders, pachygyria, spasm, subcortical-band-heterotopia.
Among the many pathways, these few ones have gauged particular interests from scientists studying X-linked Lissencephaly, and have been seen in publications frequently: Brain Development, Cell Adhesion, Cell Cycle, Cell Development, Cell Division, Cell Migration, Cognition, Cytoskeleton Organization, Localization, Mitosis, Neuroblast Proliferation, Neurogenesis, Neuron Migration, Nuclear Import, Proteolysis, Regulation Of Cell Adhesion, Translation, Transport
Quite a number of genes have been found to play important roles in X-linked Lissencephaly, such as ACACA, ARX, CALM1, CALM2, CALM3, CDK5, DCLK1, DCX, FLNA, NDEL1, PAFAH1B1, PLA2G7, PPA1, PPP1R9B, RELN, TUBA1A, UBA2, YWHAE. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.