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Disease Info Card
Intractable Epilepsy
Information about Intractable Epilepsy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Intractable Epilepsy
Most recent studies have shown that Intractable Epilepsy shares some biological mechanisms with brain-diseases, brain-neoplasms, complex-partial-seizures, cortical-dysplasia, dysplasia, epilepsies-partial, epilepsy, epilepsy-generalized, epilepsy-temporal-lobe, generalized-seizures, infantile-spasms, malformations-of-cortical-development, neoplasms, partial-seizure, sclerosis, status-epilepticus, tonic-clonic-epilepsy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Intractable Epilepsy, and have been seen in publications frequently: Brain Development, Cell Adhesion, Cell Death, Coagulation, Cognition, Drug Resistance, Excretion, Hypersensitivity, Localization, Neurogenesis, Neuroprotection, Pathogenesis, Reflex, Reverse Transcription, Rna Interference, Segmentation, Short-term Memory, Synaptic Transmission, Translation, Transport
Quite a number of genes have been found to play important roles in Intractable Epilepsy, such as ABAT, ABCB1, ABCC1, CA1, CDKL5, CSF2, GFAP, IL6, LAMC2, LRP2, MAP2, MCF2L, MTOR, POMC, SLC12A3, SLC25A5, TBC1D9, TESC, TSC1, TSC2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.