Benign Neonatal Epilepsy

Overview of Benign Neonatal Epilepsy

Most recent studies have shown that Benign Neonatal Epilepsy shares some biological mechanisms with autosomal-dominant-form-of-benign-neonatal-seizures, channelopathies, choreoathetosis, convulsion-neonatal, convulsions, dyskinetic-syndrome, encephalopathies, epilepsies-myoclonic, epilepsies-partial, epilepsy, epilepsy-frontal-lobe, epilepsy-generalized, familial-benign-neonatal-epilepsy, febrile-convulsions, infantile-spasms, myoclonic-epilepsy-juvenile, nervousness, partial-seizure, tonic-clonic-epilepsy.

Among the many pathways, these few ones have gauged particular interests from scientists studying Benign Neonatal Epilepsy, and have been seen in publications frequently: Brain Development, Coagulation, Fatty Acid Oxidation, Hypersensitivity, Localization, Neurogenesis, Pathogenesis, Pigmentation, Reflex, Segmentation, Synaptic Transmission, System Development, Transport

Quite a number of genes have been found to play important roles in Benign Neonatal Epilepsy, such as ATP1A2, CA1, CHRNA4, KCNA1, KCNQ1, KCNQ2, KCNQ3, KCNQ4, KCNQ5, POMC, PRKD1, PRRT2, PSMC1, PTGES, RPS4X, RPS4Y1, SCN1A, SCN1B, SCN2A. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Benign Neonatal Epilepsy Related Genes

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Pathways Related to Benign Neonatal Epilepsy

This information is being compiled and will come in a future update

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