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Disease Info Card
Epilepsies, Partial
Information about Epilepsies, Partial: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Epilepsies, Partial
Most recent studies have shown that Epilepsies, Partial shares some biological mechanisms with absence-epilepsy, brain-diseases, brain-neoplasms, complex-partial-seizures, convulsions, dysplasia, epilepsies-myoclonic, epilepsy, epilepsy-complex-partial, epilepsy-generalized, epilepsy-temporal-lobe, generalized-seizures, infantile-spasms, intractable-epilepsy, neoplasms, partial-seizure, sclerosis, status-epilepticus, tonic-clonic-epilepsy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Epilepsies, Partial, and have been seen in publications frequently: Aging, Brain Development, Cell Death, Coagulation, Cognition, Drug Resistance, Excretion, Hypersensitivity, Localization, Myelination, Pathogenesis, Reflex, Secretion, Segmentation, Short-term Memory, Synaptic Transmission, Translation, Transport, Visual Perception
Quite a number of genes have been found to play important roles in Epilepsies, Partial, such as AURKA, CA1, CA3, CAT, CRAT, CSF2, GLUL, GLYAT, HADHA, HTN3, LAMC2, LGALS1, LGI1, PIAS1, POMC, PRL, SCN1A, SLC25A5, SPINT2, TYMS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.