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Disease Info Card
Febrile Convulsions
Information about Febrile Convulsions: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Febrile Convulsions
Most recent studies have shown that Febrile Convulsions shares some biological mechanisms with absence-epilepsy, convulsions, encephalitis, encephalopathies, epilepsies-myoclonic, epilepsies-partial, epilepsy, epilepsy-generalized, epilepsy-temporal-lobe, infantile-severe-myoclonic-epilepsy, infective-disorder, meningitis, nervous-system-disorder, nervousness, partial-seizure, sclerosis, status-epilepticus, tonic-clonic-epilepsy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Febrile Convulsions, and have been seen in publications frequently: Aging, Brain Development, Cell Adhesion, Cell Death, Cell Migration, Coagulation, Cognition, Drug Resistance, Excretion, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Long-term Memory, Neurogenesis, Pathogenesis, Reflex, Secretion, Synaptic Transmission, Transport
Quite a number of genes have been found to play important roles in Febrile Convulsions, such as AURKA, AVP, CA1, CA3, CRP, CSF2, FST, GABRG2, GBGT1, IL1B, IL1RN, IL6, KCNQ2, LAMC2, SCN1A, SCN1B, SCN2A, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.