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- Table of Contents
Information about Myoclonic Epilepsy, Juvenile: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Myoclonic Epilepsy, Juvenile shares some biological mechanisms with absence-epilepsy, caprine-arthritis-encephalitis, convulsions, epilepsies-myoclonic, epilepsies-partial, epilepsy, epilepsy-frontal-lobe, epilepsy-generalized, epilepsy-temporal-lobe, febrile-convulsions, generalized-seizures, idiopathic-generalized-epilepsy, infantile-severe-myoclonic-epilepsy, infantile-spasms, lennox-gastaut-syndrome, myoclonus, partial-seizure, status-epilepticus, tonic-clonic-epilepsy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Myoclonic Epilepsy, Juvenile, and have been seen in publications frequently: Brain Development, Cell Adhesion, Cell Death, Cell Division, Cell Migration, Circadian Rhythm, Cognition, Drug Resistance, Hypersensitivity, Localization, M Phase, Menarche, Menstruation, Mitotic Spindle Organization, Pathogenesis, Reflex, Response To Drug, Segmentation, Short-term Memory, Spindle Organization
Quite a number of genes have been found to play important roles in Myoclonic Epilepsy, Juvenile, such as AURKA, BRD2, CHRNA4, CLCN2, CSTB, EFHC1, GABRA1, GABRG2, GJA8, ISYNA1, KCNQ2, KCNQ3, POMC, PYCARD, SCN1A, SLC27A4, TYMS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.