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Disease Info Card
Absence Epilepsy
Information about Absence Epilepsy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Absence Epilepsy
Most recent studies have shown that Absence Epilepsy shares some biological mechanisms with caprine-arthritis-encephalitis, convulsions, epilepsies-myoclonic, epilepsies-partial, epilepsy, epilepsy-generalized, epilepsy-temporal-lobe, febrile-convulsions, generalized-seizures, idiopathic-generalized-epilepsy, infantile-spasms, lennox-gastaut-syndrome, myoclonic-epilepsy-juvenile, myoclonus, partial-seizure, petit-mal-status, status-epilepticus, tonic-clonic-epilepsy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Absence Epilepsy, and have been seen in publications frequently: Aging, Brain Development, Cell Death, Circadian Rhythm, Coagulation, Cognition, Excretion, Glucose Transport, Hypersensitivity, Localization, Locomotion, Menstruation, Pathogenesis, Reflex, Secretion, Sensory Processing, Swimming, Synaptic Transmission, Translation, Transport
Quite a number of genes have been found to play important roles in Absence Epilepsy, such as CACNA1A, CACNA1H, CACNG2, CD55, CD8A, FOS, GABRG2, GJA8, MAL, POMC, SCN1A, SLC2A1, TCN2, TG, TIRAP, TNFSF14. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.