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Disease Info Card
Torsades De Pointes
Information about Torsades De Pointes: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Torsades De Pointes
Most recent studies have shown that Torsades De Pointes shares some biological mechanisms with atrial-fibrillation, atrioventricular-block, bradycardia, cardiac-arrest, cardiac-arrhythmia, cardiac-death, cardiac-fibrillation, heart-diseases, heart-failure, long-qt-syndrome, long-qt-syndrome-congenital, polymorphic-ventricular-tachycardia, premature-ventricular-contractions, sudden-cardiac-death, sudden-death, syncope, tachycardia-ventricular, ventricular-arrhythmia, ventricular-fibrillation.
Among the many pathways, these few ones have gauged particular interests from scientists studying Torsades De Pointes, and have been seen in publications frequently: Aging, Anaphylaxis, Cardiac Conduction, Excretion, Gastric Emptying, Glucose Homeostasis, Glycosylation, Hypersensitivity, Innervation, Intestinal Absorption, Localization, Pathogenesis, Platelet Aggregation, Reflex, Response To Drug, Secretion, Translation, Transport, Vasoconstriction, Vasodilation
Quite a number of genes have been found to play important roles in Torsades De Pointes, such as ACE, COL18A1, COL4A2, COLQ, CYP3A4, DCX, ERG, KCNA4, KCNA5, KCNE1, KCNE2, KCNH2, KCNH6, KCNQ1, OXSM, SCN5A, SGSM3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.