Disease Info Card

Cardiac Arrest

Information about Cardiac Arrest: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Cardiac Arrest

Most recent studies have shown that Cardiac Arrest shares some biological mechanisms with bradycardia, cardiac-arrhythmia, cardiac-death, cardiac-fibrillation, cardiopulmonary-arrest, comatose, heart-diseases, heart-failure, hemorrhage, hypotension-adverse-event, induced-hypothermia-(finding), infarction, ischemia, myocardial-infarction, out-of-hospital-cardiac-arrest, sudden-cardiac-death, sudden-death, syncope, tachycardia-ventricular, ventricular-fibrillation.

Among the many pathways, these few ones have gauged particular interests from scientists studying Cardiac Arrest, and have been seen in publications frequently: Anaphylaxis, Blood Circulation, Cardiac Conduction, Cell Death, Coagulation, Excretion, Fibrinolysis, Flight, Hemostasis, Hypersensitivity, Inflammatory Response, Localization, Neuroprotection, Pathogenesis, Reflex, Secretion, Translation, Transport, Vasoconstriction, Vasodilation

Quite a number of genes have been found to play important roles in Cardiac Arrest, such as ACE, C2, CA1, CPB1, ENO2, GLI3, GNL3, IGFALS, INS, KIF7, KRAS, NDUFB6, POR, SLC17A5, SOD1, TH, TNF, UBN1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Cardiac Arrest Related Genes

click to see detail information for each gene

ACE C2 CA1
CPB1 ENO2 GLI3
GNL3 IGFALS INS
KIF7 KRAS NDUFB6
POR SLC17A5 SOD1
TH TNF UBN1