Primary Angle-closure Glaucoma

Overview of Primary Angle-closure Glaucoma

Most recent studies have shown that Primary Angle-closure Glaucoma shares some biological mechanisms with acute-angle-closure-glaucoma, anatomical-narrow-angle-glaucoma, angle-closure-glaucoma, anterior-synechiae, blind-vision, cataract, corneal-diseases, disorder-of-the-optic-nerve, glaucoma, glaucoma-open-angle, hypertensive-disease, intraocular-pressure-disorder, ocular-hypertension, peripheral-anterior-synechiae, primary-open-angle-glaucoma, secondary-glaucoma, visual-impairment.

Among the many pathways, these few ones have gauged particular interests from scientists studying Primary Angle-closure Glaucoma, and have been seen in publications frequently: Aging, Cell Death, Circadian Rhythm, Inflammatory Response, Mating, Pathogenesis, Pigment Accumulation, Pigmentation, Reflex, Regeneration, Translation, Transport, Wound Healing

Quite a number of genes have been found to play important roles in Primary Angle-closure Glaucoma, such as ACD, APOE, ASAH1, BRCA1, CALCRL, CYP1B1, FUT2, LTA, MFRP, MMP9, MYOC, PISD, PLXNA2, PSD, RANGAP1, SLC17A5, SLC7A7, SQLE. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Primary Angle-closure Glaucoma Related Genes

click to see detail information for each gene

Pathways Related to Primary Angle-closure Glaucoma

This information is being compiled and will come in a future update

Related Diseases