Disease Info Card

Secondary Glaucoma

Information about Secondary Glaucoma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Secondary Glaucoma

Most recent studies have shown that Secondary Glaucoma shares some biological mechanisms with angle-closure-glaucoma, anterior-uveitis, blind-vision, cataract, corneal-diseases, disorder-of-eye, edema, eye-injuries, glaucoma, glaucoma-open-angle, hemorrhage, hyphema, inflammation, intraocular-pressure-disorder, melanoma, neoplasms, retinal-detachment, retinal-diseases, scleral-diseases, uveitis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Secondary Glaucoma, and have been seen in publications frequently: Aging, Angiogenesis, Cell Death, Cell Migration, Coagulation, Dehiscence, Enucleation, Hemostasis, Hypersensitivity, Inflammatory Response, Localization, Pathogenesis, Phagocytosis, Pigment Accumulation, Pigmentation, Reflex, Regeneration, Secretion, Transport, Wound Healing

Quite a number of genes have been found to play important roles in Secondary Glaucoma, such as A4GALT, ASAH1, BRCA1, CAT, CEL, CRAT, ERMAP, FAP, FBN1, FUT2, GLYAT, MYOC, PHEX, PVR, SQLE, TTR, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.