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- Table of Contents
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Disease Info Card
Glaucoma, Open-angle
Information about Glaucoma, Open-angle: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Glaucoma, Open-angle
Most recent studies have shown that Glaucoma, Open-angle shares some biological mechanisms with angle-closure-glaucoma, aqueous-humor-disorders, atrophy, blind-vision, bulla, cataract, conjunctival-diseases, corneal-diseases, disorder-of-eye, disorder-of-the-optic-nerve, exfoliation-syndrome, glaucoma, hypertensive-disease, intraocular-pressure-disorder, low-tension-glaucoma, ocular-hypertension, primary-open-angle-glaucoma, scleral-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Glaucoma, Open-angle, and have been seen in publications frequently: Aging, Blood Circulation, Cell Adhesion, Cell Death, Cell Proliferation, Coagulation, Enucleation, Innervation, Localization, Neuroprotection, Pathogenesis, Pigmentation, Reflex, Secretion, Segmentation, Senescence, Tear Secretion, Transport, Vasodilation, Wound Healing
Quite a number of genes have been found to play important roles in Glaucoma, Open-angle, such as APCS, BRCA1, CYP1B1, ERG, FN1, KCNH2, LOXL1, MYOC, OPTN, PHEX, PISD, PSD, RANGAP1, SH2D1A, SLC17A5, TNFSF14, WDR36. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.