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- Table of Contents
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Disease Info Card
Angle Closure Glaucoma
Information about Angle Closure Glaucoma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Angle Closure Glaucoma
Most recent studies have shown that Angle Closure Glaucoma shares some biological mechanisms with acute-angle-closure-glaucoma, anatomical-narrow-angle-glaucoma, aqueous-humor-disorders, blind-vision, cataract, corneal-diseases, disorder-of-eye, disorder-of-the-optic-nerve, glaucoma, glaucoma-open-angle, hypertensive-disease, intraocular-pressure-disorder, myopia, narrow-anterior-chamber-angle, ocular-hypertension, primary-angle-closure-glaucoma, primary-open-angle-glaucoma, scleral-diseases, secondary-glaucoma.
Among the many pathways, these few ones have gauged particular interests from scientists studying Angle Closure Glaucoma, and have been seen in publications frequently: Aging, Blood Circulation, Cell Death, Coagulation, Dehiscence, Enucleation, Fibroblast Proliferation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Mating, Neuroprotection, Pathogenesis, Pigmentation, Reflex, Regeneration, Secretion, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Angle Closure Glaucoma, such as ABCC6, ACD, ALPI, ASAH1, BRCA1, FOXC1, FUT2, MYOC, PHEX, PISD, PLXNA2, RANGAP1, SLC17A5, SLC7A7, SQLE, TNFSF14. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.