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Disease Info Card
Primary Open Angle Glaucoma
Information about Primary Open Angle Glaucoma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Primary Open Angle Glaucoma
Most recent studies have shown that Primary Open Angle Glaucoma shares some biological mechanisms with angle-closure-glaucoma, aqueous-humor-disorders, atrophy, blind-vision, cataract, corneal-diseases, disorder-of-the-optic-nerve, exfoliation-syndrome, glaucoma, glaucoma-open-angle, hypertensive-disease, intraocular-pressure-disorder, low-tension-glaucoma, nerve-damage, ocular-hypertension, primary-angle-closure-glaucoma, scleral-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Primary Open Angle Glaucoma, and have been seen in publications frequently: Aging, Blood Circulation, Cell Adhesion, Cell Death, Cell Proliferation, Cellular Senescence, Coagulation, Enucleation, Glycosylation, Inflammatory Response, Localization, Neuroprotection, Pathogenesis, Pigmentation, Reflex, Secretion, Senescence, Tear Secretion, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Primary Open Angle Glaucoma, such as APOE, CYP1B1, EDN1, ELN, FN1, LOXL1, MYOC, OPTN, PISD, PSD, RANGAP1, SLC17A5, TGFB2, TNF, TNFSF14, VEGFA, WDR36. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.