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Disease Info Card
Complex Partial Seizures
Information about Complex Partial Seizures: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Complex Partial Seizures
Most recent studies have shown that Complex Partial Seizures shares some biological mechanisms with absence-epilepsy, atrophy, brain-neoplasms, convulsions, depressive-disorder, epilepsies-partial, epilepsy, epilepsy-complex-partial, epilepsy-generalized, epilepsy-temporal-lobe, generalized-seizures, infantile-spasms, intractable-epilepsy, neoplasms, partial-seizure, sclerosis, simple-partial-seizures, status-epilepticus, tonic-clonic-epilepsy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Complex Partial Seizures, and have been seen in publications frequently: Aging, Cardiac Conduction, Circadian Rhythm, Cognition, Drug Resistance, Excretion, Hypersensitivity, Localization, Long-term Memory, Myelination, Neurogenesis, Ossification, Pathogenesis, Prolactin Secretion, Reflex, Response To Anticonvulsant, Secretion, Short-term Memory, Transport
Quite a number of genes have been found to play important roles in Complex Partial Seizures, such as ABAT, ACAT1, AURKA, CA1, CAT, CRAT, CSF2, EXOSC10, GFAP, GLYAT, HTN3, LAMC2, PBX1, POMC, PRL, SCN1A, SLC25A5, SPINT2, TIMM8A. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.