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Disease Info Card
Distal Muscular Dystrophies
Information about Distal Muscular Dystrophies: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Distal Muscular Dystrophies
Most recent studies have shown that Distal Muscular Dystrophies shares some biological mechanisms with atrophy, cardiomyopathies, dystrophy, hypertrophy, inclusion-body-myositis-(disorder), miyoshi-myopathy, muscle-weakness, muscular-atrophy, muscular-dystrophies-limb-girdle, muscular-dystrophy, muscular-dystrophy-duchenne, muscular-dystrophy-tibial, myopathy, myositis, myotonic-dystrophy, neuromuscular-diseases, weakness, welander-distal-myopathy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Distal Muscular Dystrophies, and have been seen in publications frequently: Autophagy, Cell Adhesion, Cell Differentiation, Chromosome Segregation, Endocytosis, Glycosylation, Inflammatory Response, Lipid Binding, Localization, Membrane Fusion, Muscle Atrophy, Muscle Contraction, Muscle Hypertrophy, Pathogenesis, Phagocytosis, Proteolysis, Regeneration, Spermatogenesis, Translation, Transport
Quite a number of genes have been found to play important roles in Distal Muscular Dystrophies, such as ANO5, CAPN3, CAV3, CHKA, CHKB, DES, DMD, DYSF, GAA, GNE, LDB3, MAPT, MATR3, MYH14, MYH7, MYOT, NEB, TTN, VCP. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.