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Disease Info Card
Inclusion Body Myositis (disorder)
Information about Inclusion Body Myositis (disorder): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Inclusion Body Myositis (disorder)
Most recent studies have shown that Inclusion Body Myositis (disorder) shares some biological mechanisms with abnormal-degeneration, alzheimers-disease, atrophy, autoimmune-diseases, autoimmune-reaction, dementia, dermatomyositis, dystrophy, inclusion-body-myopathy-sporadic, inflammation, muscle-weakness, muscular-atrophy, muscular-dystrophy, myopathy, myopathy-idiopathic-inflammatory, myositis, neuromuscular-diseases, osteitis-deformans, polymyositis, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Inclusion Body Myositis (disorder), and have been seen in publications frequently: Aging, Autophagy, Cell Activation, Cell Adhesion, Cell Death, Cell Differentiation, Cytokine Production, Glycosylation, Hyperphosphorylation, Immune Response, Inflammatory Response, Localization, Muscle Atrophy, Pathogenesis, Regeneration, Reverse Transcription, Secretion, Sensitization, Translation, Transport
Quite a number of genes have been found to play important roles in Inclusion Body Myositis (disorder), such as APOE, APP, CD8A, CHKA, CHKB, CTLA4, DES, DMPK, GEMIN4, GNE, HLA-DQA1, HLA-DRB4, HLA-E, MAPT, NOD2, PRB1, PRNP, TARDBP, TNF, VCP. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.