Disease Info Card

Welander Distal Myopathy

Information about Welander Distal Myopathy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Welander Distal Myopathy

Most recent studies have shown that Welander Distal Myopathy shares some biological mechanisms with arthrogryposis, atrophy, cardiomyopathies, distal-arthrogryposis-syndrome, distal-muscular-dystrophies, dystrophy, external-ophthalmoplegia, hypertrophic-cardiomyopathy, hypertrophy, inclusion-body-myositis-(disorder), miyoshi-myopathy, muscle-weakness, muscular-dystrophy, muscular-dystrophy-tibial, myeloproliferative-syndrome-transient, myopathy, myositis, neuromuscular-diseases, ophthalmoplegia, weakness.

Among the many pathways, these few ones have gauged particular interests from scientists studying Welander Distal Myopathy, and have been seen in publications frequently: Pathogenesis, Vasoconstriction

Quite a number of genes have been found to play important roles in Welander Distal Myopathy, such as CTLA4, DES, DMD, DYSF, GNE, HLA-DQA1, LIPG, MYH14, MYH2, MYH3, MYH7, MYH8, NCAM1, NOD2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.