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Disease Info Card
Craniofacial Dysostosis
Information about Craniofacial Dysostosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Craniofacial Dysostosis
Most recent studies have shown that Craniofacial Dysostosis shares some biological mechanisms with acrocephalosyndactylia, apert-syndrome, cleft-lip, cleft-palate, congenital-abnormal-synostosis, congenital-abnormality, craniosynostosis, cytogenetic-abnormality, dwarfism, dysostoses, dysplasia, exophthalmos, eye-abnormalities, hydrocephalus, hypoplasia, malocclusion, mandibulofacial-dysostosis, orbital-separation-excessive, strabismus.
Among the many pathways, these few ones have gauged particular interests from scientists studying Craniofacial Dysostosis, and have been seen in publications frequently: Aging, Bone Development, Bone Resorption, Brain Development, Cell Adhesion, Dehiscence, Endochondral Ossification, Localization, Mating, Ossification, Osteoblast Proliferation, Pathogenesis, Pigmentation, Secretion, Segmentation, Spermatogenesis, Tooth Eruption, Transport, Transposition, Wound Healing
Quite a number of genes have been found to play important roles in Craniofacial Dysostosis, such as ALX4, CENPJ, CSF2, CTSK, FGF13, FGF2, FGFR1, FGFR2, FGFR3, FUT3, GPSM2, HPS4, LAMC2, RUNX2, SNAI1, SS18L1, TWIST1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.