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Disease Info Card
Dysostoses
Information about Dysostoses: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Dysostoses
Most recent studies have shown that Dysostoses shares some biological mechanisms with acrodysostosis, bone-diseases, bone-diseases-developmental, cleft-palate, cleidocranial-dysplasia, congenital-abnormality, craniofacial-dysostosis, craniosynostosis, dwarfism, dysplasia, hypoplasia, jarcho-levin-syndrome, mandibulofacial-dysostosis, metaphyseal-chondrodysplasia, mucopolysaccharidoses, orbital-separation-excessive, osteochondrodysplasias, pfaundler-hurler-syndrome.
Among the many pathways, these few ones have gauged particular interests from scientists studying Dysostoses, and have been seen in publications frequently: Bone Development, Bone Remodeling, Bone Resorption, Cell Death, Dehiscence, Endochondral Ossification, Excretion, Glycosylation, Limb Development, Localization, Notch Signaling Pathway, Ossification, Pathogenesis, Secretion, Segmentation, Somitogenesis, Tooth Eruption, Transport, Transposition, Vasculogenesis
Quite a number of genes have been found to play important roles in Dysostoses, such as ARSB, CTSK, DLL3, FGFR2, FUT3, GLB1, HHIP, HPS4, LFNG, MESP2, MTSS1, REG3A, RPL29, SCD, ST13, TCOF1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.