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Disease Info Card
Orbital Separation Excessive
Information about Orbital Separation Excessive: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Orbital Separation Excessive
Most recent studies have shown that Orbital Separation Excessive shares some biological mechanisms with blepharoptosis, cleft-lip, cleft-palate, congenital-abnormality, congenital-cerebral-hernia, congenital-heart-defects, craniofacial-abnormalities, craniofacial-dysostosis, craniosynostosis, cytogenetic-abnormality, developmental-delay-(disorder), dwarfism, dysplasia, hypoplasia, hypospadias, low-set-ears, microcephaly, micrognathism, trisomy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Orbital Separation Excessive, and have been seen in publications frequently: Bone Maturation, Cell Division, Cell Proliferation, Dna Methylation, Excretion, Interphase, Limb Development, Localization, Mating, Meiosis, Metaphase, Methylation, Ossification, Pathogenesis, Pigmentation, Reflex, Segmentation, Translation, Transport, Transposition
Quite a number of genes have been found to play important roles in Orbital Separation Excessive, such as CDK5R1, CSF2, EFNB1, FGFR2, FGFR3, GLI3, INVS, LAMC2, LMLN, MB, MID1, MPG, PTPN11, SUB1, TGFBR1, TPPP, TWIST1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.