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Disease Info Card
Complete Hydatidiform Mole
Information about Complete Hydatidiform Mole: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Complete Hydatidiform Mole
Most recent studies have shown that Complete Hydatidiform Mole shares some biological mechanisms with bleeding-of-vagina, choriocarcinoma, gestational-trophoblastic-neoplasms, hemorrhage, hydatidiform-mole, hydatidiform-mole-invasive, hyperplasia, malignant-neoplasms, malignant-paraganglionic-neoplasm, multiple-pregnancy, neoplasm-metastasis, neoplasms, partial-hydatidiform-mole, pre-eclampsia, pregnancy-complications-neoplastic, triploidy, trophoblastic-neoplasms, trophoblastic-tumor-placental-site, uterine-neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Complete Hydatidiform Mole, and have been seen in publications frequently: Cell Adhesion, Cell Cycle, Cell Proliferation, Cytokinesis, Dna Amplification, Dna Methylation, Fertilization, Immune Response, Interphase, Localization, Meiosis, Meiosis I, Methylation, Mismatch Repair, Oogenesis, Ovulation, Pathogenesis, Reverse Transcription, Vasculogenesis
Quite a number of genes have been found to play important roles in Complete Hydatidiform Mole, such as CD34, CDKN1C, CHM, CIB2, CORO1A, CSH1, CSH2, EGFR, ERBB2, IGF2, LNPEP, MPZ, PAM, PCNA, PHLDA2, PRB1, TP53. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.