This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Trophoblastic Tumor, Placental Site
Information about Trophoblastic Tumor, Placental Site: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Trophoblastic Tumor, Placental Site
Most recent studies have shown that Trophoblastic Tumor, Placental Site shares some biological mechanisms with bleeding-of-vagina, carcinoma, cell-invasion, choriocarcinoma, complete-hydatidiform-mole, epithelioid-trophoblastic-tumor, exaggerated-placental-site, gestational-trophoblastic-neoplasms, hemorrhage, hydatidiform-mole, hydatidiform-mole-invasive, lung-neoplasms, malignant-neoplasms, malignant-paraganglionic-neoplasm, neoplasm-metastasis, neoplasms, partial-hydatidiform-mole, trophoblastic-neoplasms, uterine-neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Trophoblastic Tumor, Placental Site, and have been seen in publications frequently: Angiogenesis, Cell Adhesion, Cell Cycle, Cell Differentiation, Cell Growth, Cell Proliferation, Coagulation, Drug Resistance, Fertilization, Localization, Luteinization, Meiosis, Meiosis I, Menopause, Menstruation, Mitosis, Pathogenesis, Secretion, Transport
Quite a number of genes have been found to play important roles in Trophoblastic Tumor, Placental Site, such as CDKN1C, CKAP4, CSH1, CSH2, CTLA4, DBI, EGFR, EPO, ETFA, HLA-DQA1, HLA-G, MCAM, MUC1, NOD2, NR0B1, RPE65, SLC22A4, TP53, UVRAG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.