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- Table of Contents
Information about Partial Hydatidiform Mole: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Partial Hydatidiform Mole shares some biological mechanisms with choriocarcinoma, complete-hydatidiform-mole, gestational-trophoblastic-neoplasms, hemorrhage, hydatidiform-mole, hydatidiform-mole-invasive, hyperplasia, malignant-paraganglionic-neoplasm, neoplasm-metastasis, neoplasms, polyploidy, pre-eclampsia, pregnancy-complications, pregnancy-complications-neoplastic, tetraploidy, triploidy, trisomy, trophoblastic-neoplasms, uterine-neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Partial Hydatidiform Mole, and have been seen in publications frequently: Cell Cycle, Cell Division, Cell Proliferation, Dna Methylation, Dosage Compensation, Fertilization, Immune Response, Insemination, Interphase, Localization, Meiosis, Meiosis I, Meiosis Ii, Menstruation, Methylation, Mismatch Repair, Ovulation, Pathogenesis, Secretion, Vasculogenesis
Quite a number of genes have been found to play important roles in Partial Hydatidiform Mole, such as AFP, CDKN1C, CGA, CHM, CIB2, CORO1A, CSH1, CSH2, ERBB2, MPZ, NR0B1, PAM, PCNA, PHLDA2, PRB1, TP53. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.