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- Table of Contents
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Disease Info Card
Low Tension Glaucoma
Information about Low Tension Glaucoma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Low Tension Glaucoma
Most recent studies have shown that Low Tension Glaucoma shares some biological mechanisms with angle-closure-glaucoma, atrophy, blind-vision, cataract, corneal-diseases, disorder-of-the-optic-nerve, exfoliation-syndrome, glaucoma, glaucoma-open-angle, hemorrhage, hypertensive-disease, intraocular-pressure-disorder, ischemia, migraine-disorders, nerve-damage, ocular-hypertension, optic-atrophy, primary-open-angle-glaucoma, vasospasm.
Among the many pathways, these few ones have gauged particular interests from scientists studying Low Tension Glaucoma, and have been seen in publications frequently: Aging, Blood Circulation, Cell Death, Coagulation, Fibrinolysis, Immune Response, Localization, Myelination, Neuroprotection, Pathogenesis, Pigment Accumulation, Pigmentation, Platelet Aggregation, Reflex, Response To Cold, Secretion, Swimming, Transport, Vasoconstriction, Vasodilation
Quite a number of genes have been found to play important roles in Low Tension Glaucoma, such as APCS, CTSD, DCT, EDN1, LUC7L3, MED12, MYOC, OPA1, OPTN, PISD, PSD, SH2D1A, SLC1A3, TNF, TP53, WDR36. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.