Disease Info Card

Exfoliation Syndrome

Information about Exfoliation Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Exfoliation Syndrome

Most recent studies have shown that Exfoliation Syndrome shares some biological mechanisms with angle-closure-glaucoma, aqueous-humor-disorders, cataract, conjunctival-diseases, corneal-diseases, diabetes-mellitus, disorder-of-eye, disorder-of-the-optic-nerve, glaucoma, glaucoma-open-angle, hypertensive-disease, inflammation, intraocular-lymphoma, intraocular-pressure-disorder, intraoperative-complications, lens-diseases, mydriasis, ocular-hypertension, pigment-dispersion-syndrome-(disorder), primary-open-angle-glaucoma.

Among the many pathways, these few ones have gauged particular interests from scientists studying Exfoliation Syndrome, and have been seen in publications frequently: Aging, Coagulation, Complement Activation, Dehiscence, Delamination, Enucleation, Inflammatory Response, Innervation, Localization, Muscle Contraction, Pathogenesis, Pigment Accumulation, Pigmentation, Protein Oxidation, Reflex, Secretion, Tear Secretion, Translation, Transport, Wound Healing

Quite a number of genes have been found to play important roles in Exfoliation Syndrome, such as AP3B1, B3GAT1, BRCA1, CLU, ELN, LOXL1, MTHFR, MYOC, PEX19, PHEX, PREP, PTGDS, SLC26A4, TIMP1, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.