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Disease Info Card
Optic Nerve Injuries
Information about Optic Nerve Injuries: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Optic Nerve Injuries
Most recent studies have shown that Optic Nerve Injuries shares some biological mechanisms with blind-vision, brain-injuries, craniocerebral-trauma, crushing-injury, disorder-of-the-optic-nerve, edema, eye-injuries, fracture, glaucoma, hemorrhage, intraocular-pressure-disorder, nerve-damage, nerve-degeneration, nerve-transections-optic, nervousness, nonpenetrating-wounds, orbital-fractures, skull-fractures, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Optic Nerve Injuries, and have been seen in publications frequently: Axon Regeneration, Axonogenesis, Cell Adhesion, Cell Death, Cell Proliferation, Cellular Localization, Enucleation, Immune Response, Inflammatory Response, Innervation, Localization, Macrophage Activation, Myelination, Neuroprotection, Pathogenesis, Phagocytosis, Reflex, Regeneration, Secretion, Transport
Quite a number of genes have been found to play important roles in Optic Nerve Injuries, such as BCL2, BDNF, CASP3, CAT, CNTF, CRAT, ERG, ERMAP, FGF2, GCA, GCLC, GDNF, GFAP, GLUL, GLYAT, JUN, KCNH2, MAPK3, NTF3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.