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Disease Info Card
Crushing Injury
Information about Crushing Injury: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Crushing Injury
Most recent studies have shown that Crushing Injury shares some biological mechanisms with compression-of-spinal-cord, crush-syndrome, crushing-sensation, edema, finger-injuries, fracture, hemorrhage, inflammation, ischemia, kidney-failure, nerve-damage, nerve-degeneration, nervousness, nonpenetrating-wounds, optic-nerve-injuries, pain, peripheral-nerve-injuries, sciatic-neuropathy, spinal-cord-injuries, wallerian-degeneration.
Among the many pathways, these few ones have gauged particular interests from scientists studying Crushing Injury, and have been seen in publications frequently: Aging, Axon Regeneration, Cell Death, Cell Proliferation, Coagulation, Diuresis, Hypersensitivity, Inflammatory Response, Innervation, Localization, Myelination, Neuroprotection, Pathogenesis, Phagocytosis, Reflex, Regeneration, Schwann Cell Proliferation, Secretion, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Crushing Injury, such as BDNF, CLIP1, CLIP2, FGF2, GDNF, GFAP, IL6, NGF, NGFR, NOS1, NOS2, NTF3, POMC, RPL5, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.