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Disease Info Card
Nerve Transections, Optic
Information about Nerve Transections, Optic: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Nerve Transections, Optic
Most recent studies have shown that Nerve Transections, Optic shares some biological mechanisms with albinism, blind-vision, disorder-of-the-optic-nerve, glaucoma, hypertensive-disease, inflammation, intraocular-pressure-disorder, ischemia, nerve-damage, nerve-degeneration, nervousness, neurodegenerative-disorders, ocular-hypertension, optic-nerve-injuries, retinal-degeneration, retinal-diseases, retrograde-degeneration, tissue-adhesions, wallerian-degeneration.
Among the many pathways, these few ones have gauged particular interests from scientists studying Nerve Transections, Optic, and have been seen in publications frequently: Axon Regeneration, Cell Adhesion, Cell Cycle, Cell Death, Cell Proliferation, Cellular Localization, Coagulation, Enucleation, Hatching, Induction Of Apoptosis, Innervation, Localization, Myelination, Neuroprotection, Pathogenesis, Programmed Cell Death, Proteolysis, Reflex, Regeneration, Transport
Quite a number of genes have been found to play important roles in Nerve Transections, Optic, such as ADCYAP1, BCL2, BCL2L1, BDNF, CASP3, CNTF, ERG, ERMAP, GCA, GCLC, GDNF, GFAP, GLUL, GMCL1, JUN, KCNH2, NTF3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.