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Disease Info Card
Craniocerebral Trauma
Information about Craniocerebral Trauma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Craniocerebral Trauma
Most recent studies have shown that Craniocerebral Trauma shares some biological mechanisms with brain-concussion, brain-edema, brain-injuries, cerebral-hemorrhage, closed-head-injuries, comatose, contusions, edema, epilepsy, fracture, headache, hematoma, hematoma-subdural, hemorrhage, intracranial-hypertension, multiple-trauma, nonpenetrating-wounds, skull-fractures, traumatic-brain-injury.
Among the many pathways, these few ones have gauged particular interests from scientists studying Craniocerebral Trauma, and have been seen in publications frequently: Aging, Blood Coagulation, Cell Death, Coagulation, Cognition, Excretion, Flight, Hemostasis, Immune Response, Inflammatory Response, Localization, Neuroprotection, Ossification, Pathogenesis, Reflex, Regeneration, Secretion, Translation, Transport, Vasoconstriction
Quite a number of genes have been found to play important roles in Craniocerebral Trauma, such as ALB, APOE, AR, ASRGL1, AVP, CAT, CRAT, CSF2, ENO2, F2, GCLC, GLYAT, IL6, LAMC2, NLRP5, POMC, S100B, TNF, TP63, UGCG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.