This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Nonconvulsive Status Epilepticus
Information about Nonconvulsive Status Epilepticus: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Nonconvulsive Status Epilepticus
Most recent studies have shown that Nonconvulsive Status Epilepticus shares some biological mechanisms with absence-epilepsy, brain-injuries, comatose, complex-partial-status-epilepticus, confusion, convulsions, encephalitis, encephalopathies, epilepsies-partial, epilepsy, epilepsy-complex-partial, epilepsy-generalized, grand-mal-status-epilepticus, impaired-cognition, partial-seizure, petit-mal-status, status-epilepticus, tonic-clonic-epilepsy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Nonconvulsive Status Epilepticus, and have been seen in publications frequently: Aging, Cell Death, Cognition, Conjugation, Hypersensitivity, Localization, Menopause, Metaphase, Neuroprotection, Pathogenesis, Reflex, Response To Anticonvulsant, Sensory Processing, Social Behavior, Swimming, Transport
Quite a number of genes have been found to play important roles in Nonconvulsive Status Epilepticus, such as AURKA, CA1, CHRNA4, CSF2, ENO1, FUT2, GCLC, GFAP, KCNQ2, LAMC2, NOTCH3, NOTCH4, PRNP, PRPH2, SH3BP4, SLC17A5, SQLE, UGCG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.