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Disease Info Card
Optic Atrophy
Information about Optic Atrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Optic Atrophy
Most recent studies have shown that Optic Atrophy shares some biological mechanisms with ataxia, atrophy, blind-vision, cataract, complete-hearing-loss, diabetes-mellitus, disorder-of-eye, disorder-of-the-optic-nerve, edema, glaucoma, intraocular-pressure-disorder, neoplasms, optic-atrophies-hereditary, optic-atrophy-autosomal-dominant, optic-atrophy-hereditary-leber, optic-neuritis, papilledema, retinal-diseases, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Optic Atrophy, and have been seen in publications frequently: Aging, Cell Death, Coagulation, Dna Repair, Enucleation, Excretion, Insulin Secretion, Localization, Membrane Fusion, Mitochondrial Fission, Mitochondrial Fusion, Myelination, Neuroprotection, Oxidative Phosphorylation, Pathogenesis, Pigmentation, Reflex, Secretion, Translation, Transport
Quite a number of genes have been found to play important roles in Optic Atrophy, such as CSF2, CYCS, DNM1L, ERG, INS, KCNH2, LAMC2, MED12, MFN1, MFN2, OPA1, OPA3, PMEL, POMC, TNFSF14, WFS1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.