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Disease Info Card
Iris Atrophy
Information about Iris Atrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Iris Atrophy
Most recent studies have shown that Iris Atrophy shares some biological mechanisms with anterior-uveitis, atrophy, cataract, chandler-syndrome, cogan-reese-syndrome, corneal-diseases, corneal-edema, disorder-of-eye, edema, glaucoma, inflammation, intraocular-pressure-disorder, iridocorneal-endothelial-syndrome, iridocyclitis, iris-diseases, progressive-iris-atrophy, secondary-glaucoma, uveal-diseases, uveitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Iris Atrophy, and have been seen in publications frequently: Blood Circulation, Cell Death, Cell Migration, Coagulation, Contact Inhibition, Dedifferentiation, Enucleation, Immune Response, Neuroprotection, Pathogenesis, Pigmentation, Reflex, Secretion, Tear Secretion, Viral Replication
Quite a number of genes have been found to play important roles in Iris Atrophy, such as BRCA1, C1QTNF5, CASP1, CES2, DCN, EPYC, FGF2, FOXC1, GJA1, LUM, NOL3, PITX2, RPS19, TYRP1, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.