This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
Information about Iridocorneal Endothelial Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Iridocorneal Endothelial Syndrome shares some biological mechanisms with anterior-synechiae, atrophy, cataract, chandler-syndrome, cogan-reese-syndrome, corneal-diseases, corneal-edema, dystrophy, edema, fuchs-endothelial-dystrophy, glaucoma, hereditary-corneal-dystrophy, intraocular-pressure-disorder, iris-atrophy, iris-diseases, melanocytic-nevus, peripheral-anterior-synechiae, progressive-iris-atrophy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Iridocorneal Endothelial Syndrome, and have been seen in publications frequently: Aging, Enucleation, Localization, Pathogenesis, Pigmentation, Tissue Remodeling, Wound Healing
Quite a number of genes have been found to play important roles in Iridocorneal Endothelial Syndrome, such as ALPK3, BRCA1, CALM2, CASP1, CES2, CS, DES, DMPK, MUC1, RFC1, RFC2, VIM, VSX1, ZEB1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.
Aging | Enucleation | Localization |
Pathogenesis | Pigmentation | Tissue Remodeling |
Wound Healing |