pathway Info Card

Contact Inhibition

Information about Contact Inhibition: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Contact Inhibition

Most recent studies have shown that Contact Inhibition shares some biological mechanisms with angiogenesis, cell-adhesion, cell-cell-adhesion, cell-cycle, cell-cycle-arrest, cell-death, cell-differentiation, cell-division, cell-growth, cell-migration, cell-motility, cell-proliferation, localization, locomotion, pathogenesis, s-phase, secretion, senescence, transport, wound-healing.

Among the many pathways, these few ones have gauged particular interests from scientists studying Contact Inhibition, and have been seen in publications frequently: angiogenesis, cell-adhesion, cell-cell-adhesion, cell-cycle, cell-cycle-arrest, cell-death, cell-differentiation, cell-division, cell-growth, cell-migration, cell-motility, cell-proliferation, localization, locomotion, pathogenesis, s-phase, secretion, senescence, transport, wound-healing

Quite a number of genes have been found to play important roles in Contact Inhibition, such as CDH1, CDK2, CDK4, CDKN1A, CDKN1B, CDKN2A, DCTN6, EGF, FN1, IFI27, MAPK1, MYC, NF2, PSMD9, RB1, TMED7, TP53. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this pathway. Plesae stay updated.

Contact Inhibition Related Genes

click to see detail information for each gene

CDH1 CDK2 CDK4
CDKN1A CDKN1B CDKN2A
DCTN6 EGF FN1
IFI27 MAPK1 MYC
NF2 PSMD9 RB1
TMED7 TP53