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Disease Info Card
Iris Diseases
Information about Iris Diseases: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Iris Diseases
Most recent studies have shown that Iris Diseases shares some biological mechanisms with angle-closure-glaucoma, atrophy, blepharoptosis, cataract, corneal-diseases, disorder-of-eye, glaucoma, glaucoma-open-angle, intraocular-pressure-disorder, intraoperative-complications, iridocorneal-endothelial-syndrome, melanoma, neoplasms, retinal-detachment, retinal-diseases, tissue-adhesions, uveal-diseases, uveal-neoplasms, uveitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Iris Diseases, and have been seen in publications frequently: Aging, Cell Adhesion, Cell Migration, Cell Proliferation, Coagulation, Dehiscence, Enucleation, Hypersensitivity, Immune Response, Inflammatory Response, Innervation, Localization, Muscle Contraction, Neural Crest Cell Migration, Pathogenesis, Pigment Accumulation, Pigmentation, Reflex, Regeneration, Smooth Muscle Contraction
Quite a number of genes have been found to play important roles in Iris Diseases, such as ASAH1, BRCA1, CASP1, CAT, CES2, CRAT, GLYAT, GPNMB, SS18L1, TNFSF14, TYR, TYRP1, VEGFA, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.